DISORDER, PMS, INFERTILITY, ENDOMETRIOSIS, DYSMENORRHEA
of menstrual disorders include Dysmenorrhea (severe
menstrual cramps), Amenorrhea (absence of menstruation),
Menorrhagia (heavy bleeding), Premenstrual syndrome
(PMS), Endometriosis (displacement of uterine
tissue outside the uterus), and Infertility.
IUCCA UPPER CERVICAL CARE RELATES TO MENSTRUAL
women, six key hormones serve as chemical messengers
that regulate the reproductive system. The hypothalamus
first releases the gonadotropin-releasing hormone
(GnRH). This chemical, in turn, stimulates the
pituitary gland to produce follicle-stimulating
hormone (FSH) and luteinizing hormone (LH). Estrogen,
progesterone, and the male hormone testosterone
are secreted by the ovaries at the command of
FSH and LH and complete the hormonal group necessary
for reproductive health.
Evidence supports that certain cases of menstrual
disorders result from the malfunction of nerves
in the brain and spinal cord (neuroendocrine system)
that control the production of hormones. The abnormal
hormone levels can be due to a malfunction between
the brain, hypothalamus, pituitary, spinal cord,
and ovaries, and can be head/neck trauma-induced.1-24
purpose of IUCCA upper cervical care is to reverse
the trauma-induced neck injury; thereby reducing
irritation to the injured nerves involved in the
neuroendocrine system. While many menstrual disorder
sufferers recall specific traumas
such as head injuries, auto accidents or falls
that preceded the onset of their symptoms, some
do not. An upper cervical examination utilizing
Radiography and Digital
Infrared Imaging is
necessary in each individual's case to assess
whether an upper cervical injury is present and
whether benefit from IUCCA upper cervical care
can be achieved.
Disorders Case Studies (PMS, Infertility, Endometriosis, Dysmenorrhea)
1. Berezin M, Ohry A, Shemesh Y. Hyperprolactinemia,
galactorrhea and amenorrhea in women with a spinal
cord injury. Gynecol Endocrinol 1989 Jun; 3(2):
2. Massol J, Humbert O, Cattin F. Post-traumatic
diabetes insipidus and amenorrhea-galactorrhea
syndrome after pituitary stalk rupture. Neuroradiology
1987; 29(3): 299-300.
3. Joele LJ, Endtz LJ. Traumatic disorders of
pituitary-hypothalamic function. Excretion of
follicle-stimulating hormone after closed head
injury. A preliminary investigation. Appl Neurophysiol
1975; 38(2): 110-4.
4. Rotkina IE. Gonadotropic function of the pituitary
gland in women in the acute period of craniocerebral
injury. Probl Endokrinol Gormonoter 1966 Jul-Aug;
12 (4): 41-5.
5. Grossman WF, Sanfield JA. Hypothalamic atrophy
presenting as amenorrhea and sexual infantilism
in a female adolescent. A case report. J Reprod
Med 1994 Sep; 39(9):738-40.
6. Girard J, Marelli R. Posttraumatic hypothalamo-pituitary
insufficiency. J Pediatr 1977 Feb; 90(2): 241-2.
7. Della Corte F, Mancini A, Valle D. Provocative
hypothalamopituitary axis tests in severe head
injury: correlations with severity and prognosis.
Crit Care Med 1998 Aug; 26(8): 1419-26.
8. Childrers MK, Rupright J, Jones PS. Assessment
of neuroendocrine dysfunction following traumatic
brain injury. Brain Inj 1998 Jun; 12(6): 517-23.
9. Iglesias P, Gomez-Pan A, Diez JJ. Spontaneous
recovery from post-traumatic hypopituitarism.
J Endocrinol Invest 1996 May; 19(5): 320-3.
10. Chiolero R, Berger M. Endocrine response to
brain injury. New Horiz 1994 Nov; 2(4):432-42.
11. Woolf PD. Hormonal responses to trauma. Crit
Care Med 1992 Feb; 20(2): 216-26.
12. Gunn IR, Beastall GH, Matthews DM. Post-traumatic
hypothalamic-pituitary dysfunction presenting
with biochemical features of primary hypothyroidism.
Ann Clin Biochem 1991 Jul; 28(Pt 4):327-30.
13. Lim HS, Ang BK, Ngim RC. Hypopituitarism following
head injury-a case report. Ann Acad Med Singapore
1990 Nov; 19(6): 851-5.
14. Barton RN. The neuroendocrinology of physical
injury. Baillieres Clin Endocrinol Metab 1987;
15. King LR, Knowles HC Jr, McLaurin RL. Pituitary
hormone response to head injury. Neurosurgery
1981 Sep; 9(3):229-35.
16. Kosteljanetz M, Jensen TS, Norgard B. Sexual
and hypothalamic dysfunction in the postconcussional
syndrome. Acta Neurol Scand 1981 Mar; 63(3): 169-80.
17. Shutov AA, Chudinov AA, Pleshkova NM. State
of the hypothalamo-hypophyseo-thyroid system in
severe craniocerebral injuries. Zh Vopr Neirokhir
Im N N Burdenko 1980 May-Jun; (3): 23-7.
18. Valenta LJ, De Feo DR. Post-traumatic hypopituitarism
due to a hypothalamic lesion. Am J Med 1980 Apr;
19. Landau H, Adin I, Spitz IM. Pituitary insufficiency
following head injury. Isr J Med Sci 1978 Jul;
20. Rudman D, Fleischer AS, Kutner MH. Suprahypophyseal
hypogonadism and hypothyroidism during prolonged
coma after head trauma. J Clin Endocrinol Metab
1977 Oct; 45(4): 747-54.
21. Winternitz WW, Dzur JA. Pituitary failure
secondary to head trauma. Case report. J Neurosurg
1976 Apr; 44(4): 504-5.
22. Wick, G., et al. Immunoendocrine Communication
via The Hypothalamus-Pituitary-Adrenal Axis in
Autoimmune Diseases. Endocrine Reviews. 14:539-563,
23. Denckla WD. Interactions between age and the
neuroendocrine and immune systems. Fed Proc 1978;37:1263-1267
24. Korr IM. Sustained sympathecotonia as a factor
in disease. In: Korr IM, ed. The neurobiological
mechanisms in manipulative therapy. New York:
Plenum, 1978 229-268.
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