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Case Studies: Thyroid Disorders, Hypothyroidism, Hyperthyroidism

IUCCA Upper Cervical


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Symptoms of hypothyroidism (underactive thyroid) include: fatigue, weakness, cold hands and feet, low body temperature, dry sparse hair, dry skin, weight gain despite loss of appetite, poor memory, low sex drive, slow pulse, low blood pressure, and menstrual irregularities, to name a few. Symptoms of hyperthyroidism (overactive thyroid) include: weight loss, increased appetite, restlessness, nervousness, irritability, overactivity, increased heart rate, heart palpitations, increased sweating, tremors, shakiness, double vision, and protrusion of eyes.


Evidence supports that certain cases of thyroid disorders result from the malfunction of nerves in the brain and spinal cord that control the thyroid's production of thyroid hormone. Whether too little or too much thyroid hormone is produced, the abnormal level can be due to a malfunction between the brain, pituitary, spinal cord, and thyroid, and can be head/neck trauma-induced.1-24

The purpose of IUCCA upper cervical care is to reverse the trauma-induced neck injury; thereby reducing irritation to the injured nerves that supply the thyroid. While many thyroid disease sufferers recall specific traumas such as head injuries, auto accidents or falls that preceded the onset of their symptoms, some do not. An upper cervical examination utilizing Laser-aligned Radiography and Digital Infrared Imaging is necessary in each individual's case to assess whether an upper cervical injury is present and whether benefit from IUCCA upper cervical care can be achieved.


Thyroid Disorders Case Studies (Hypothyroidism, Hyperthyroidism)

1. Sehnert KW, Croft AC. Basal metabolic temperature vs. laboratory assessment in posttraumatic hypothyroidism. J Manipulative Physiol Ther 1996 Jan; 19(1): 6-12.
2. Girard J, Marelli R. Posttraumatic hypothalamo-pituitary insufficiency. J Pediatr 1977 Feb; 90(2): 241-2.
3. Hwang SL, Lieu AS, Howng SL. Hypothalamic dysfunction in acute head-injured patients with stress ulcer. Kaohsiung J Med Sci 1998 Sep; 14((): 554-60.
4. Della Corte F, Mancini A, Valle D. Provocative hypothalamopituitary axis tests in severe head injury: correlations with severity and prognosis. Crit Care Med 1998 Aug; 26(8): 1419-26.
5. Childrers MK, Rupright J, Jones PS. Assessment of neuroendocrine dysfunction following traumatic brain injury. Brain Inj 1998 Jun; 12(6): 517-23.
6. Iglesias P, Gomez-Pan A, Diez JJ. Spontaneous recovery from post-traumatic hypopituitarism. J Endocrinol Invest 1996 May; 19(5): 320-3.
7. Chiolero R, Berger M. Endocrine response to brain injury. New Horiz 1994 Nov; 2(4):432-42.
8. Murav'ev OB, Maiorova EM, Volkov AV. Characteristics of changes in the thyroid status in severe trauma and massive hemorrhage. Anesteziol Reanimatol 1994 Sep-Oct; (5): 14-7
.9. Woolf PD. Hormonal responses to trauma. Crit Care Med 1992 Feb; 20(2): 216-26.
10. Gunn IR, Beastall GH, Matthews DM. Post-traumatic hypothalamic-pituitary dysfunction presenting with biochemical features of primary hypothyroidism. Ann Clin Biochem 1991 Jul; 28(Pt 4):327-30.
11. Lim HS, Ang BK, Ngim RC. Hypopituitarism following head injury-a case report. Ann Acad Med Singapore 1990 Nov; 19(6): 851-5.
12. Shutov AA, Chudinov AA. Disorders of thyroid function as a consequence of severe craniocerebral injury. Zh Nevropatol Psikhiatr Im S S Korsakova 1988; 88(5): 11-3.
13. Chiolero RL, Lemarchand-Beraud T, Schutz Y. Thyroid function in severely traumatized patients with or without head injury. Acta Endocrinol 1988 Jan; 117(1):80-6.
14. Barton RN. The neuroendocrinology of physical injury. Baillieres Clin Endocrinol Metab 1987; 1(2): 355-74.
15. King LR, Knowles HC Jr, McLaurin RL. Pituitary hormone response to head injury. Neurosurgery 1981 Sep; 9(3):229-35.
16. Kosteljanetz M, Jensen TS, Norgard B. Sexual and hypothalamic dysfunction in the postconcussional syndrome. Acta Neurol Scand 1981 Mar; 63(3): 169-80.
17. Shutov AA, Chudinov AA, Pleshkova NM. State of the hypothalamo-hypophyseo-thyroid system in severe craniocerebral injuries. Zh Vopr Neirokhir Im N N Burdenko 1980 May-Jun; (3): 23-7.
18. Valenta LJ, De Feo DR. Post-traumatic hypopituitarism due to a hypothalamic lesion. Am J Med 1980 Apr; 68(4):614-7.
19. Landau H, Adin I, Spitz IM. Pituitary insufficiency following head injury. Isr J Med Sci 1978 Jul; 14(7): 785-9.
20. Rudman D, Fleischer AS, Kutner MH. Suprahypophyseal hypogonadism and hypothyroidism during prolonged coma after head trauma. J Clin Endocrinol Metab 1977 Oct; 45(4): 747-54.
21. Winternitz WW, Dzur JA. Pituitary failure secondary to head trauma. Case report. J Neurosurg 1976 Apr; 44(4): 504-5.
22. Wick, G., et al. Immunoendocrine Communication via The Hypothalamus-Pituitary-Adrenal Axis in Autoimmune Diseases. Endocrine Reviews. 14:539-563, October 1993.
23. Denckla WD. Interactions between age and the neuroendocrine and immune systems. Fed Proc 1978;37:1263-1267
24. Korr IM. Sustained sympathecotonia as a factor in disease. In: Korr IM, ed. The neurobiological mechanisms in manipulative therapy. New York: Plenum, 1978 229-268.


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